This site is intended for U.S. residents only

Bylvay helps reduce reuptake of bile acids (BAs)1,2

All images are actor portrayals.

MOA

A selective, reversible IBAT inhibitor at a key site of enterohepatic circulation1,2

Role of IBAT3

  • The IBAT regulates BA pool size, with 95% of BA reabsorbed by the IBAT

By targeting the IBAT, Bylvay2,3*

  • Blocks reabsorption of BAs and reroutes them via fecal excretion
  • Helps reduce the BA pool recirculating to the liver

*Although the complete mechanism of Bylvay is unknown, it may involve inhibition of the IBAT, which results in decreased reuptake of bile salts, as observed by a decrease in sBA.1

MOA

Bylvay is selective, with minimal systemic exposure1,3

  • Bylvay acts locally in the small intestine
  • Bylvay is minimally absorbed in plasma following oral administration, and no accumulation of Bylvay was observed following once-daily dosing
  • Highly efficient IBAT inhibition at a starting dose of 40 mcg/kg/day in PFIC or 120 mcg/kg/day in PFIC or 120 mcg/kg/day in ALGS

See how Bylvay works to reduce all levels of cholestatic pruritus

WATCH VIDEO
Video image

ALGS=Alagille syndrome; BA=bile acid; IBAT=ileal bile acid transporter; PFIC=progressive familial intrahepatic cholestasis; sBA=serum bile acid.

References:

  1. Bylvay Prescribing Information. Boston, MA: Albireo Pharma, Inc.; 2023.
  2. Kamath BM, Stein P, Houwen RHJ, Verkade HJ. Potential of ileal bile acid transporter inhibition as a therapeutic target in Alagille syndrome and progressive familial intrahepatic cholestasis. Liver Int. 2020;40(8):1812-1822.
  3. Data on file A4250-005. Boston, MA: Albireo Pharma, Inc.
  1. Bylvay Prescribing Information. Boston, MA: Albireo Pharma, Inc.; 2023.
  2. Kamath BM, Stein P, Houwen RHJ, Verkade HJ. 
    Potential of ileal bile acid transporter inhibition as a
    therapeutic target in Alagille syndrome and
    progressive familial intrahepatic cholestasis. Liver Int.
    2020;40(8):1812-1822.
  3. Data on file A4250-005. Boston, MA: Albireo Pharma,
    Inc.